Metabolic strugGLS after FLT3 inhibition in AML

Research output: Contribution to journalReview articlepeer-review

2 Scopus citations


In this issue of Blood, Gallipoli et al report that by performing an elegant clustered regularly interspaced short palindromic repeats (CRISPR) screen of FLT3 internal tandem duplication–positive (FLT3-ITD1) acute myeloid leukemia (AML) cells, they have identified that FLT3 inhibition exposes a therapeutically relevant metabolic dependency on glutaminolysis.

Original languageEnglish (US)
Pages (from-to)1631-1632
Number of pages2
Issue number15
StatePublished - Apr 12 2018

ASJC Scopus subject areas

  • Biochemistry
  • Immunology
  • Hematology
  • Cell Biology


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