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Interferon alpha: The key trigger of type 1 diabetes
Angela Lombardi
, Effie Tsomos
, Sara S. Hammerstad
, Yaron Tomer
Medicine
Research output
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Contribution to journal
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Review article
›
peer-review
78
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Keyphrases
Interferon-α (IFN-α)
100%
Type I Diabetes
100%
Type 1 Diabetes Mellitus (T1DM)
100%
T Cells
50%
Novel Therapeutics
50%
Infected Tissue
50%
Viral Infection
50%
Innate Immune Response
50%
B Cells
50%
Innate Immunity
50%
Monocytes
50%
Islet
50%
Beneficial Effects
50%
Systemic Lupus Erythematosus
50%
Chemokines
50%
Rheumatoid Arthritis
50%
Endoplasmic Reticulum Stress (ER stress)
50%
Autoimmune Disease
50%
Natural Killer Cells
50%
Autoimmunity
50%
Autoantigen
50%
Cell Activation
50%
Mitochondrial Function
50%
Adaptive Immunity
50%
Pathophysiological Mechanisms
50%
Susceptible Individuals
50%
Autoreactive
50%
HLA-I
50%
Priming Mechanisms
50%
Primary Biliary Cholangitis
50%
Autoimmune Thyroid Disease
50%
Insulin Production
50%
CD8 T Lymphocytes
50%
β-cell Death
50%
Immunology and Microbiology
Cytokine
100%
T Cell
100%
Innate Immune System
100%
Interferon Alfa
100%
Secretion (Process)
50%
Adaptive Immune System
50%
CD8
50%
Natural Killer Cell
50%
Cell Death
50%
Monocyte
50%
Autoimmunity
50%
Autoimmune Disease
50%
Systemic Lupus Erythematosus
50%
Chemokine
50%
Inflammatory Arthritis
50%
Autoantigen
50%
Cell Activation
50%
Endoplasmic Reticulum Stress
50%
Autoimmune Thyroiditis
50%
Primary Biliary Cirrhosis
50%
Insulin
50%
Virus Infection
50%