Abstract
We have accumulated a significant body of evidence that support the hypothesis that aPL antibody-mediated disruption of annexin A5 crystallization is a mechanism for RSPL in the APS. Further investigation of the functions of the annexins in placental physiology and disease are likely to be productive areas for future research.
Original language | English (US) |
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Pages (from-to) | 77-81 |
Number of pages | 5 |
Journal | Thrombosis Research |
Volume | 115 |
Issue number | SUPPL. |
State | Published - Feb 2005 |
ASJC Scopus subject areas
- Hematology