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Cholecystectomy: A way forward and back to metabolic syndrome?
Gabriella Garruti
, David Q.H. Wang
, Agostino Di Ciaula
, Piero Portincasa
Research output
:
Contribution to journal
›
Review article
›
peer-review
32
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Scopus citations
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Keyphrases
Bile Acids
100%
Metabolic Syndrome
100%
Cholecystectomy
100%
Liver
33%
Digestion
33%
Energy Expenditure
33%
Intestine
33%
Lipid Homeostasis
33%
Gallbladder
33%
Nuclear Receptor
33%
Fibroblast Growth Factor 19 (FGF19)
33%
Brown Fat
33%
Glucos
33%
Dietary Lipids
33%
Gallstone Patients
33%
Asymptomatic Gallstones
33%
Bile Acid Receptor
33%
Enterohepatic Recycling
33%
Gallbladder Removal
33%
Independent Risk Factors
33%
Metabolic Pathways
33%
Risk Factors for Development
33%
Receptors, G-protein-coupled
33%
Farnesoid
33%
Medicine and Dentistry
Metabolic Syndrome
100%
Cholecystectomy
100%
Bile Acid
100%
Fat Intake
25%
Secretion (Process)
25%
Energy Expenditure
25%
Receptor
25%
Gallstone
25%
Cell Nucleus Receptor
25%
Enterohepatic Circulation
25%
G Protein Coupled Receptor
25%
Fibroblast Growth Factor 19
25%
Brown Adipose Tissue
25%
Lipid Homeostasis
25%
Metabolic Pathway
25%
Pharmacology, Toxicology and Pharmaceutical Science
Bile Acid
100%
Metabolic Syndrome X
100%
Lipid
66%
G Protein Coupled Receptor
33%
Gallstone
33%
Fibroblast Growth Factor 19
33%
Cell Nucleus Receptor
33%
G Protein-Coupled Bile Acid Receptor
33%
Biochemistry, Genetics and Molecular Biology
Bile Acid
100%
Secretion (Process)
33%
G Protein Coupled Receptor
33%
Lipid Homeostasis
33%
Nuclear Receptor
33%
Enterohepatic Circulation
33%
Energy Expenditure
33%
FGF19
33%
G Protein-Coupled Bile Acid Receptor
33%
Lipid
33%