B7x/B7-H4 modulates the adaptive immune response and ameliorates renal injury in antibody-mediated nephritis

Keyphrases

• Adaptive Immune Response 100%
• Macrophages 100%
• Antibody-mediated 100%
• Renal Injury 100%
• B7-H4 100%
• Nephritis 100%
• Kidney 66%
• T Cells 66%
• Autoimmune Disease 66%
• Kidney Injury 66%
• Treatment Options 33%
• Cause of Death 33%
• Spleen 33%
• Current Treatment 33%
• Infiltration 33%
• Novel Therapeutics 33%
• Lupus 33%
• Lupus Nephritis 33%
• Immunoglobulin 33%
• Local Infiltration 33%
• Inflammatory Mediators 33%
• Inflammation Injury 33%
• Kidney Disease 33%
• Non-lymphoid Tissues 33%
• Tissue Injury 33%
• B7 Family 33%
• CD4 T Cells 33%
• Passive Administration 33%
• Intrinsic Renal Cells 33%
• Local Inflammation 33%
• Functional Response 33%
• Co-inhibitory Molecules 33%
• Proliferation Response 33%
• Target Needs 33%
• Inflammatory Phenotype 33%
• Robust Adaptive 33%
• CD8 T Cells 33%
• Anti-GBM Disease 33%
• Autoimmune Kidney Disease 33%

Immunology and Microbiology

• Macrophage 100%
• Adaptive Immune System 100%
• Nephritis 100%
• T Cell 66%
• Autoimmune Disease 66%
• Lupus Erythematosus 66%
• Inflammation 66%
• Cytotoxic T-Cell 33%
• Mediator 33%
• Intravenous Immunoglobulin 33%
• T-Helper Cell 33%
• Glomerulonephritis 33%
• Upregulation 33%
• Antigen 33%
• Lymphoid Tissue 33%