Abstract
Understanding how genetic risk variants contribute to complex diseases is crucial for predicting disease susceptibility and developing patient-tailored therapies. In this issue of Cell Stem Cell, Young et al. (2015) dissect the function of common non-coding risk haplotypes in the SORL1 locus in the pathogenesis of sporadic Alzheimer's disease using patient-derived induced pluripotent stem cells.
Original language | English (US) |
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Pages (from-to) | 341-342 |
Number of pages | 2 |
Journal | Cell Stem Cell |
Volume | 16 |
Issue number | 4 |
DOIs | |
State | Published - Apr 2 2015 |
Externally published | Yes |
ASJC Scopus subject areas
- Molecular Medicine
- Genetics
- Cell Biology