Dissecting risk haplotypes in sporadic Alzheimer's disease

Frank Soldner, Rudolf Jaenisch

Research output: Contribution to journalShort surveypeer-review

7 Scopus citations

Abstract

Understanding how genetic risk variants contribute to complex diseases is crucial for predicting disease susceptibility and developing patient-tailored therapies. In this issue of Cell Stem Cell, Young et al. (2015) dissect the function of common non-coding risk haplotypes in the SORL1 locus in the pathogenesis of sporadic Alzheimer's disease using patient-derived induced pluripotent stem cells.

Original languageEnglish (US)
Pages (from-to)341-342
Number of pages2
JournalCell Stem Cell
Volume16
Issue number4
DOIs
StatePublished - Apr 2 2015
Externally publishedYes

ASJC Scopus subject areas

  • Molecular Medicine
  • Genetics
  • Cell Biology

Fingerprint

Dive into the research topics of 'Dissecting risk haplotypes in sporadic Alzheimer's disease'. Together they form a unique fingerprint.

Cite this